January 1-4, 2026 | Location to be Determined
Scientific Organizers:
January 1-4, 2026 | Location to be Determined
Scientific Organizers:
Important Deadlines
Meeting Summary
Autophagy dysregulation has been linked to a wide range of diseases, such as cancer, neurodegenerative disorders, metabolic disorders, and infections. However, further research is needed to fully understand the precise roles and mechanisms of autophagy in different disease contexts and its therapeutic potential. This meeting aims to address gaps in knowledge regarding the regulation, mechanisms, and therapeutic potential of selective autophagy in disease contexts. A further understanding of selective autophagy is critical for developing targeted therapeutic interventions and understanding disease mechanisms, as selective autophagy allows cells to specifically target and remove damaged organelles, misfolded proteins, and intracellular pathogens. This meeting will showcase ongoing basic research advances in the field, but also focus on translational research and industry developments in the area. A joint meeting with “Membrane Dynamics, Repair and Disease” is an excellent opportunity for engaging joint sessions of shared interest, in particular with the pathology. We also plan to organize a workshop on tools and strategies to study and target autophagy, which may also include lessons learned from clinical trials. The translational sessions in this meeting provide a forum for exchange of knowledge between academic and industry researchers which is not an element in on-going meetings such as Gordon Research conferences, or the International Symposium for Autophagy.
SPECIAL RECOGNITION:
KEYSTONE SYMPOSIA THANKS OUR SPONSOR(S) FOR GENEROUSLY SUPPORTING THIS MEETING:
KEYSTONE SYMPOSIA THANKS OUR MEETING EXHIBITOR(S):
KEYSTONE SYMPOSIA THANKS THESE DONOR(S) FOR GENEROUSLY SUPPORTING THIS MEETING:
THESE COMPANIES HAVE GENEROUSLY AGREED TO COVER THE EXPENSES OF THEIR EMPLOYEES WHO ARE SPEAKING AT THIS MEETING:
GRANT RECOGNITION:
KEYSTONE SYMPOSIA THANKS OUR GIFT-IN-KIND MEDIA SPONSORS
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