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This meeting took place in 2004
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Molecular Control of Adipogenesis and Obesity (X2)
Organizer(s) Stephen R. Farmer and Sheila Collins
March 4—10, 2004
Fairmont Banff Springs • Banff, Alberta Canada
Abstract Deadline: Nov 3, 2003
Late Abstract Deadline:
Scholarship Deadline:
Early Registration Deadline: Jan 5, 2004
Sponsored by Abbott Laboratories and Biovitrum AB
Summary of Meeting:
The focus of this conference is on understanding the molecular mechanisms that control the formation and function of adipose tissue as well as understanding how various physiologic processes acting through adipose tissue regulate metabolism and energy homeostasis. This is a very timely conference since there has been an alarming increase in obesity and its associated disorders (most notably type 2 diabetes) throughout the United States and the Western World during the last 10 years. It is clear now that obesity is associated with an increased risk of diabetes. From 1991 to 2000, there was a 61% increase in obesity and 49% increase in type 2 diabetes among adults of both genders. The most exciting current research in this field includes: 1. Identification of the adipocyte as an endocrine cell which acts at the center of a complex physiologic process to control overall metabolism and energy balance by secreting hormones, cytokines and other peptides that regulate functions in the hypothalamus, muscle and liver. 2. Characterization of adipocyte-specific secreted proteins such as adiponectin that act on the liver and muscle to sensitize these tissues to insulin. 3. Continued on-going research on leptin - defining its mechanism of action within the brain and in the periphery. 4. The role of circulating fatty acids and their accumulation in peripheral tissues such as liver and muscle where they contribute to the insulin resistance observed in these tissues in obese individuals. 5. Identification and characterization of the signaling pathways and transcription factors that regulate the formation of adipose tissue (adipogenesis) and thermogenesis in adipocytes and muscle cells. 6. Elucidation of the role of nuclear hormone receptors and their associated cofactors in regulating glucose and lipid homeostasis. In addition, several of these receptors are targets for drug discovery to combat obesity and to sensitize individuals to insulin. This is most elegantly illustrated by the elucidation of the mechanism of action of the thiazolidinedione family of insulin sensitizers, which are ligands for PPAR gamma (the master regulator of adipogenesis). Some of the major problems are: Understanding the contribution of genetics versus the environment and life style to obesity and type 2 diabetes. Awaiting bioinformatics from the genome and proteome progects to help elucidate some of these problems. A further understanding of the integration of insulin-responsive tissues in regulating metabolism and energy balance. To identify new hormones and peptides that mediate the signaling processes among these different tissues. To determine why visceral fat depots are linked to a higher risk of cardiovascular disease and diabetes in obese individuals. The goal of this meeting is to bring together experts in the listed areas of research to present their lastest results so that the obesity and diabetes research community can integrate this information with their own observations to begin to understand the underlying cause of obesity and its related disorders. This meeting is strengthened by the fact that it is a joint meeting with Diabetes Mellitus: Molecular Mechanisms, Genetics and New Therapies, which will facilitate an even greater interaction across additional fields of expertise.
View Scholarships/Awards
The focus of this conference is on understanding the molecular mechanisms that control the formation and function of adipose tissue as well as understanding how various physiologic processes acting through adipose tissue regulate metabolism and energy homeostasis. This is a very timely conference since there has been an alarming increase in obesity and its associated disorders (most notably type 2 diabetes) throughout the United States and the Western World during the last 10 years. It is clear now that obesity is associated with an increased risk of diabetes. From 1991 to 2000, there was a 61% increase in obesity and 49% increase in type 2 diabetes among adults of both genders. The most exciting current research in this field includes: 1. Identification of the adipocyte as an endocrine cell which acts at the center of a complex physiologic process to control overall metabolism and energy balance by secreting hormones, cytokines and other peptides that regulate functions in the hypothalamus, muscle and liver. 2. Characterization of adipocyte-specific secreted proteins such as adiponectin that act on the liver and muscle to sensitize these tissues to insulin. 3. Continued on-going research on leptin - defining its mechanism of action within the brain and in the periphery. 4. The role of circulating fatty acids and their accumulation in peripheral tissues such as liver and muscle where they contribute to the insulin resistance observed in these tissues in obese individuals. 5. Identification and characterization of the signaling pathways and transcription factors that regulate the formation of adipose tissue (adipogenesis) and thermogenesis in adipocytes and muscle cells. 6. Elucidation of the role of nuclear hormone receptors and their associated cofactors in regulating glucose and lipid homeostasis. In addition, several of these receptors are targets for drug discovery to combat obesity and to sensitize individuals to insulin. This is most elegantly illustrated by the elucidation of the mechanism of action of the thiazolidinedione family of insulin sensitizers, which are ligands for PPAR gamma (the master regulator of adipogenesis). Some of the major problems are: Understanding the contribution of genetics versus the environment and life style to obesity and type 2 diabetes. Awaiting bioinformatics from the genome and proteome progects to help elucidate some of these problems. A further understanding of the integration of insulin-responsive tissues in regulating metabolism and energy balance. To identify new hormones and peptides that mediate the signaling processes among these different tissues. To determine why visceral fat depots are linked to a higher risk of cardiovascular disease and diabetes in obese individuals. The goal of this meeting is to bring together experts in the listed areas of research to present their lastest results so that the obesity and diabetes research community can integrate this information with their own observations to begin to understand the underlying cause of obesity and its related disorders. This meeting is strengthened by the fact that it is a joint meeting with Diabetes Mellitus: Molecular Mechanisms, Genetics and New Therapies, which will facilitate an even greater interaction across additional fields of expertise.
View Scholarships/Awards
No registration fees are used to fund entertainment or alcohol at this conference
THURSDAY, MARCH 4
FRIDAY, MARCH 5
SATURDAY, MARCH 6
SUNDAY, MARCH 7
MONDAY, MARCH 8
TUESDAY, MARCH 9
WEDNESDAY, MARCH 10
Conference Program Print | View meeting in 12 hr (am/pm) time
THURSDAY, MARCH 4
19:30—21:30
Keynote Session (Joint)
Meeting has ended...abstracts no longer viewable online.
*
Michael P. Czech,
University of Massachusetts Medical School, USA
Allen M. Spiegel,
National Institutes of Health, USA
The NIDDK Agenda for Support of Diabetes and Obesity Research
The NIDDK Agenda for Support of Diabetes and Obesity Research
Stephen W. Fesik,
Vanderbilt University School of Medicine, USA
RNAi for Target Identification and Validation
RNAi for Target Identification and Validation
08:00—11:15
The Adipocyte as an Endocrine Cell: Regulation of Regulation of Energy Metabolism, Brain, Muscle, and Liver and Beta Cells (Joint)
Meeting has ended...abstracts no longer viewable online.
Jeffrey M. Friedman,
Rockefeller University, USA
Leptin and the Neural Circuit Regulating Weight and Food Intakeand the Adaptive Response to Starvation - Rapid Rewiring of the Arcute Neuclus
Leptin and the Neural Circuit Regulating Weight and Food Intakeand the Adaptive Response to Starvation - Rapid Rewiring of the Arcute Neuclus
Barbara B. Kahn,
Beth Israel Deaconess Medical Center, Harvard Medical School, USA
AMPK Regulates Food Intake by Responding to Leptin and Other Hormonal Signals and Nutrients in the Hypothalmus
AMPK Regulates Food Intake by Responding to Leptin and Other Hormonal Signals and Nutrients in the Hypothalmus
Takashi Kadowaki,
University of Tokyo, Japan
Adiponectin Receptors: Structure, Function and Signal Transduction Mechanism
Adiponectin Receptors: Structure, Function and Signal Transduction Mechanism
Jeffrey Flier,
Harvard University, USA
The Role of SOCS3 in Leptin and Insulin Action
The Role of SOCS3 in Leptin and Insulin Action
Grant A. Mitchell,
CHU Sainte-Justine, Canada
Short Talk: Hormone-Sensitive Lipase (HSL)-Independent Adipocyte Lipolysis during Beta Adrenergic Stimulation, Fasting and Dietary Fat Loading
Short Talk: Hormone-Sensitive Lipase (HSL)-Independent Adipocyte Lipolysis during Beta Adrenergic Stimulation, Fasting and Dietary Fat Loading
E. Dale Abel,
University of Iowa, Carver College of Medicine, USA
Short Talk: Mechanism by which Insulin Resistance Leads to Mitochondrial Dysfunction in Cardiac Muscle
Short Talk: Mechanism by which Insulin Resistance Leads to Mitochondrial Dysfunction in Cardiac Muscle
16:45—19:00
Adipogenesis I: Signaling and Transcription Factors
Meeting has ended...abstracts no longer viewable online.
*
Stephen R. Farmer,
Boston University School of Medicine, USA
Ormond A. MacDougald,
University of Michigan, USA
Role of Wnt Signaling in Development of Adipose Tissues and Bone
Role of Wnt Signaling in Development of Adipose Tissues and Bone
Jacqueline M. Stephens,
Louisiana State University, USA
Crosstalk between STATs and PPARgamma in Adipocytes
Crosstalk between STATs and PPARgamma in Adipocytes
Evan D. Rosen,
Harvard University, USA
Transcriptional Pathways in Adipose-Selective Gene Expression
Transcriptional Pathways in Adipose-Selective Gene Expression
16:45—19:00
Regulated Secretory Pathways in Adipocytes and Beta Cells
Meeting has ended...abstracts no longer viewable online.
*
Amira Klip,
Hospital for Sick Children, Canada
Timothy E. McGraw,
Weill Medical College of Cornell University, USA
GLUT4 Trafficking Pathways
GLUT4 Trafficking Pathways
Christopher J. Rhodes,
MedImmune, USA
Regulation of Insulin Exocytosis beyond Generating Secondary Signals
Regulation of Insulin Exocytosis beyond Generating Secondary Signals
Peter A. Antinozzi,
Wake Forest School of Medicine, USA
Short Talk: A RNAi-Based Genetic Screen of Mitochondrial Enzymes Identified Pyruvate Carboxylase as an Essential Gene in Facilitating Glucose-Stimulated Insulin Secretion
Short Talk: A RNAi-Based Genetic Screen of Mitochondrial Enzymes Identified Pyruvate Carboxylase as an Essential Gene in Facilitating Glucose-Stimulated Insulin Secretion
Ruth E. Gimeno,
Eli Lilly and Company, USA
Short Talk: Deletion of FATP5 Reveals a Role for Bile Acid Recycling in Body Weight Homeostasis
Short Talk: Deletion of FATP5 Reveals a Role for Bile Acid Recycling in Body Weight Homeostasis
08:00—11:00
Stem Cells and Development of Insulin Responsive Tissues (Joint)
Meeting has ended...abstracts no longer viewable online.
*
Douglas A. Melton,
Harvard University, USA
Stem Cells for Pancreatic Development
Stem Cells for Pancreatic Development
Chris Wright,
Vanderbilt University Medical Center, USA
Pancreas/Forecut Develoment - Response to PDX1 Gene Dosage
Pancreas/Forecut Develoment - Response to PDX1 Gene Dosage
Ronald M. Evans,
HHMI/The Salk Institute, USA
Nuclear Receptors and the Complex Journey to Obesity
Nuclear Receptors and the Complex Journey to Obesity
Duncan Odom,
University of Cambridge, UK
Short Talk: A Broad Role for HNF4alpha in Pancreatic Islet Transcription
Short Talk: A Broad Role for HNF4alpha in Pancreatic Islet Transcription
Donalyn Scheuner,
University of Michigan, USA
Short Talk: Translational Control is Required to Prevent High-Fat Diet-Induced Diabetes and Obesity
Short Talk: Translational Control is Required to Prevent High-Fat Diet-Induced Diabetes and Obesity
14:00—16:00
Hot Topics 1
Bernard Binétruy,
INSERM, France
Mice Lacking ERK1 Have Less Fat and are Protected from Diet-Induced Obesity
Mice Lacking ERK1 Have Less Fat and are Protected from Diet-Induced Obesity
Paul S. Cooke,
University of Illinois at Urbana-Champaign, USA
Knockout of the Cell Cycle Regulators p27Kip1 and/or p21Cip1 Produces Adipocyte Hyperplasia and Obesity
Knockout of the Cell Cycle Regulators p27Kip1 and/or p21Cip1 Produces Adipocyte Hyperplasia and Obesity
Melina Fan,
Addgene, USA
p160MBP Repression of PGCI in the Absence of p38 Activity
p160MBP Repression of PGCI in the Absence of p38 Activity
Jacob E. Friedman,
University of Oklahoma Health Sciences Center, USA
Deletion of CCAT/Enhancer Binding Protein beta (C/EBP beta) Attenuates Diabetes and Steatotosis in Leprdb/db Mice
Deletion of CCAT/Enhancer Binding Protein beta (C/EBP beta) Attenuates Diabetes and Steatotosis in Leprdb/db Mice
Kira R. Gantt,
East Carolina University Brody School of Medicine, USA
HuR Binds and Shuttles C/EBP-beta mRNA to the Cytosol, An Early Event in Adipogenesis
HuR Binds and Shuttles C/EBP-beta mRNA to the Cytosol, An Early Event in Adipogenesis
Justin J. Rochford,
University of Aberdeen, UK
ETO is a Novel Inhibitor of C/EBPbeta Involved in the Hormonal Control of Adipogenesis
ETO is a Novel Inhibitor of C/EBPbeta Involved in the Hormonal Control of Adipogenesis
Robert L. Kortum,
National Cancer Institute, National Institutes of Health, USA
The Molecular Scaffold Kinase Suppressor of Ras 1 (KSR1) Regulates ERK Activation and Determines Adipogenic Potential
The Molecular Scaffold Kinase Suppressor of Ras 1 (KSR1) Regulates ERK Activation and Determines Adipogenic Potential
Chih-Hao Lee,
Harvard University School of Public Health, USA
PPAR delta Facilitates VLDL-Derived Fatty Acid Catabolism in the Macrophage
PPAR delta Facilitates VLDL-Derived Fatty Acid Catabolism in the Macrophage
16:45—19:00
Adipogenesis II: Chromatin and Coregulators
Meeting has ended...abstracts no longer viewable online.
Robert G. Roeder,
Rockefeller University, USA
Function of Diverse PPARgamma Coactivators in Adipogenesis
Function of Diverse PPARgamma Coactivators in Adipogenesis
Anthony N. Imbalzano,
University of Massachusetts Medical School, USA
Activation of PPARgamma2 Expression during Adipogenesis
Activation of PPARgamma2 Expression during Adipogenesis
*
Johan Auwerx,
École Polytechnique Fédérale de Lausanne, Switzerland
Cofactors and Nuclear Receptors in the Control of Adipometabolism
Cofactors and Nuclear Receptors in the Control of Adipometabolism
Mark Christian,
Imperial College London, UK
Short Talk: RIP140 Co-Repressor is a Key Regulator of Adipose Biology
Short Talk: RIP140 Co-Repressor is a Key Regulator of Adipose Biology
16:45—19:15
Signaling and Secretion Networks
Meeting has ended...abstracts no longer viewable online.
*
Yoshitomo Oka,
Tohoku University Graduate School of Medicine, Japan
David Ron,
University of Cambridge, UK
Stress Signal Integration by Phosphorylated Translation Initiation Factor 2 (eIF2alpha)
Stress Signal Integration by Phosphorylated Translation Initiation Factor 2 (eIF2alpha)
Morris F. White,
Boston Children's Hospital, Harvard Medical School, USA
Coordinate Regulation by the IRS Proteins
Coordinate Regulation by the IRS Proteins
Michael P. Czech,
University of Massachusetts Medical School, USA
Analysis of Insulin Signaling through RNAi-based Gene Silencing
Analysis of Insulin Signaling through RNAi-based Gene Silencing
Mark W. Sleeman,
Monash University, Australia
Short Talk: Identification and Characterization of Novel Genes Associated with Obesity/Diabetes by a High-Throughput KO Process, VelociGene®
Short Talk: Identification and Characterization of Novel Genes Associated with Obesity/Diabetes by a High-Throughput KO Process, VelociGene®
08:00—11:00
PPARs, Orphan Receptors and Metabolism
Meeting has ended...abstracts no longer viewable online.
Andrea L. Hevener,
University of California, Los Angeles, USA
Role of Skeletal Muscle and Adipocyte PPARgamma Receptors in Insulin Sensitivity in Thiazoladinedione Action
Role of Skeletal Muscle and Adipocyte PPARgamma Receptors in Insulin Sensitivity in Thiazoladinedione Action
*
Barbara C. Hansen,
University of South Florida, Tampa, USA
PPAR’s and their Calorie-Restriction Mimetic Effects in Non Human Primates
PPAR’s and their Calorie-Restriction Mimetic Effects in Non Human Primates
Yaacov Barak,
Magee-Womens Research Institute, USA
Studies of Adipogenesis and Insulin Resistance by Genetically Manipulating PPAR gamma
Studies of Adipogenesis and Insulin Resistance by Genetically Manipulating PPAR gamma
Stephen R. Farmer,
Boston University School of Medicine, USA
Signaling Pathways Regulating PPARgamma Activity during Adipogenesis
Signaling Pathways Regulating PPARgamma Activity during Adipogenesis
08:00—09:30
Insulin Signaling to GLUT4: Breaking Stories
Meeting has ended...abstracts no longer viewable online.
*
Jeffrey E. Pessin,
Albert Einstein College of Medicine, USA
Konstantin V. Kandror,
Boston University School of Medicine, USA
Formation of the Insulin-Responsive Vesicles in 3T3-L1 Adipocytes
Formation of the Insulin-Responsive Vesicles in 3T3-L1 Adipocytes
Jonathan S. Bogan,
Yale University School of Medicine, USA
Molecular Regulation of GLUT4 Trafficking
Molecular Regulation of GLUT4 Trafficking
09:50—11:15
Panel Discussion: PI3-Kinase-Dependent and -Independent Paths for Insulin-Regulated GLUT4 Translocation? At what steps is it Required?
Alan R. Saltiel,
University of California, San Diego, USA
CAP/Cb1 TC10 Pathway
CAP/Cb1 TC10 Pathway
Jeremy M. Tavaré,
University of Bristol, UK
Glut4 Translocation/Trafficking
Glut4 Translocation/Trafficking
Paul F. Pilch,
Boston University School of Medicine, USA
Lipid Microdomains IR Receptor and Caveolin
Lipid Microdomains IR Receptor and Caveolin
David E. James,
University of Sydney, Australia
Insulin Regulation of Glucose Transport by Akt-Dependent and Independent Pathways
Insulin Regulation of Glucose Transport by Akt-Dependent and Independent Pathways
Amira Klip,
Hospital for Sick Children, Canada
PI3-Kinase Inputs in GLUT4 Compartmentalization and Membrane Fusion
PI3-Kinase Inputs in GLUT4 Compartmentalization and Membrane Fusion
Yannick Le Marchand-Brustel,
INSERM, France
Pathway for Osmotic Stress Induced Glucose Uptake
Pathway for Osmotic Stress Induced Glucose Uptake
Jeffrey E. Pessin,
Albert Einstein College of Medicine, USA
Insulin Stimulated Trafficking
Insulin Stimulated Trafficking
14:00—16:00
Hot Topics 2
Hei Sook Sul,
University of California, Berkeley, USA
ADSF/Resistin Inhibits Adipogenesis but Improves Insulin Sensitivity
ADSF/Resistin Inhibits Adipogenesis but Improves Insulin Sensitivity
Claire M. Steppan,
University of Pennsylvania, USA
SOCS3 is a Potential Mediator of Resistin Antagonism of Insulin Action
SOCS3 is a Potential Mediator of Resistin Antagonism of Insulin Action
Nathan E. Wolins,
Washington University, USA
Adipocytes Remodel to Accommodate Rapid Triacylglycerol Synthesis and Packaging
Adipocytes Remodel to Accommodate Rapid Triacylglycerol Synthesis and Packaging
Stuart P. Weisberg,
Columbia University, USA
Obesity is Associated with Macrophage Accumulation in Adipose Tissue
Obesity is Associated with Macrophage Accumulation in Adipose Tissue
Kristoffer Ström,
Lund University, Sweden
Investigations into the Resistance to Diet-Induced Obesity and Adipose Tissue Insulin Resistance of Hormone-Sensitive Lipase Deficient Mice
Investigations into the Resistance to Diet-Induced Obesity and Adipose Tissue Insulin Resistance of Hormone-Sensitive Lipase Deficient Mice
Femke Streijger,
Universitair Medisch Centrum, St. Radboud, NCMLS, Netherlands
Mice Deficient for Both Brain-Type Creatine Kinases have Smaller White Adipocytes and A Reduced Lipid Content in Brown Adipocytes
Mice Deficient for Both Brain-Type Creatine Kinases have Smaller White Adipocytes and A Reduced Lipid Content in Brown Adipocytes
Marc R. Van Gilst,
Fred Hutchinson Cancer Research Center, USA
Nuclear Hormone Receptors in C. Elegans
Nuclear Hormone Receptors in C. Elegans
Peteris Alberts,
Biovitrum, Sweden
The 11beta-Hydroxysteroid Dehydrogenase Type 1 Inhibitor BVT.2733 has Beneficial Effects in Three Mouse Models of Obesity and Diabetes, Including DIO Mice
The 11beta-Hydroxysteroid Dehydrogenase Type 1 Inhibitor BVT.2733 has Beneficial Effects in Three Mouse Models of Obesity and Diabetes, Including DIO Mice
16:45—19:00
Signaling, Nuclear Factors and Glucose Homeostasis (Joint)
Meeting has ended...abstracts no longer viewable online.
*
Richard W. Hanson,
Case Western Reserve University School of Medicine, USA
Vamsi K. Mootha,
Massachusetts General Hospital, USA
Short Talk: Genomics Approaches to Human Diabetes
Short Talk: Genomics Approaches to Human Diabetes
Roger J. Davis,
HHMI/University of Massachusetts Medical School, USA
Signal Transduction by Stress-Activated MAP Kinases
Signal Transduction by Stress-Activated MAP Kinases
Domenico Accili,
Columbia University, USA
Regulation of Insulin-Dependent Gene Expression by the fFrkhead Transcription Factor Foxo1
Regulation of Insulin-Dependent Gene Expression by the fFrkhead Transcription Factor Foxo1
Gary B. Ruvkun,
Massachusetts General Hospital, Harvard Medical School, USA
Fat Storage in C. Elegans
Fat Storage in C. Elegans
08:00—11:00
Genes and Signals Controlling White and Brown Adipocyte Function
Meeting has ended...abstracts no longer viewable online.
*
Sheila Collins,
Vanderbilt University Medical Center, USA
Components of the PKA -> p38 MAPK Signaling Cascade in Adipocytes
Components of the PKA -> p38 MAPK Signaling Cascade in Adipocytes
Leslie P. Kozak,
Polish Academy of Sciences, Poland
Genetic Variation in Regulatory Pathways Associated with Brown Adipocyte Induction
Genetic Variation in Regulatory Pathways Associated with Brown Adipocyte Induction
Iichiro Shimomura,
Osaka University Graduate School of Medicine, Japan
New Molecular Target of Obesity-Related Metabolic Syndrome
New Molecular Target of Obesity-Related Metabolic Syndrome
Karsten Kristiansen,
University of Copenhagen, Denmark
The Retinoblastoma Protein is a Regulator of White Versus Brown Adipocyte Differentiation
The Retinoblastoma Protein is a Regulator of White Versus Brown Adipocyte Differentiation
08:00—11:15
Lessons from Expressed Gene and Proteome Databases
Meeting has ended...abstracts no longer viewable online.
Klaus H. Kaestner,
University of Pennsylvania School of Medicine, USA
Functional Genomics of the Beta-Cell: From Libraries to Targets
Functional Genomics of the Beta-Cell: From Libraries to Targets
Silvia Corvera,
University of Massachusetts Medical School, USA
Proteomic Analysis of TZD Actions
Proteomic Analysis of TZD Actions
Alan D. Attie,
University of Wisconsin-Madison, USA
Genetic and Genomic Dissection of Diabetes in Mice
Genetic and Genomic Dissection of Diabetes in Mice
Yoshitomo Oka,
Tohoku University Graduate School of Medicine, Japan
A Role of Wolfram Syndrome Gene in Beta Cell Apoptosis and Stimulus-Secretion Coupling
A Role of Wolfram Syndrome Gene in Beta Cell Apoptosis and Stimulus-Secretion Coupling
Xiaoli Chen,
National Institutes of Health, USA
Short Talk: Identification of Insulin-Regulated Secretory Proteins in Rat Adipose Cells and their Relevance to Insulin Resistance
Short Talk: Identification of Insulin-Regulated Secretory Proteins in Rat Adipose Cells and their Relevance to Insulin Resistance
Marie S. Thearle,
NIH/NIDDK/PECRB, USA
Short Talk: Body Mass-Hepatic Quantitative Trait Transcripts - A Common Set of Hepatic Transcripts Regulated by Body Mass
Short Talk: Body Mass-Hepatic Quantitative Trait Transcripts - A Common Set of Hepatic Transcripts Regulated by Body Mass
16:45—19:30
Diabetes and Obesity Genes (Joint)
Meeting has ended...abstracts no longer viewable online.
*
Masato Kasuga,
National Center for Global Health and Medicine, Japan
Vijay K. Yechoor,
University of Pittsburgh, USA
Genetic Analysis of Insulin and Diabetes Regulated Gene Expression
Genetic Analysis of Insulin and Diabetes Regulated Gene Expression
T. Keith Blackwell,
Joslin Diabetes Center and Harvard Medical School, USA
Short Talk: Regulation of a Conserved Oxidative Stress Defense in C. elegans
Short Talk: Regulation of a Conserved Oxidative Stress Defense in C. elegans
08:00—11:00
Control of Lipid Metabolism. Session Sponsored in part by Lipomics Technologies, Inc.
Meeting has ended...abstracts no longer viewable online.
*
Rosalind A. Coleman,
University of North Carolina at Chapel Hill, USA
Regulation of Triacylglycerol Biosynthesis by GPAT
Regulation of Triacylglycerol Biosynthesis by GPAT
Karen Reue,
University of California, Los Angeles, USA
Lipin, A Modulator of Adiposity
Lipin, A Modulator of Adiposity
08:00—11:15
Role of Cytokines in Type II Diabetes and Insulin Resistance
Meeting has ended...abstracts no longer viewable online.
*
André Marette,
Laval University Hospital Research Center, Canada
Steven E. Shoelson,
Harvard Medical School, Joslin Diabetes Center, USA
Inflammation in Insulin Resistance: Pathogenic Mediator and Target for Reversal
Inflammation in Insulin Resistance: Pathogenic Mediator and Target for Reversal
Gökhan S. Hotamisligil,
Harvard School of Public Health, USA
Crossroads between Inflammatory and Metabolic Pathways in Diabetes
Crossroads between Inflammatory and Metabolic Pathways in Diabetes
D. Grahame Hardie,
University of Dundee, UK
The LKBI —> AMPK Pathway: A Key Target for Anti-Diabetic Drugs
The LKBI —> AMPK Pathway: A Key Target for Anti-Diabetic Drugs
Roger H. Unger,
University of Texas Southwestern Medical Center, USA
Lipotoxic Diseases and their Prevention by Adipocytokines
Lipotoxic Diseases and their Prevention by Adipocytokines
Matthias Hundt,
La Jolla Institute for Allergy and Immunology, USA
Short Talk: Increased Insulin Sensitivity in Mice Lacking Protein Kinase C theta
Short Talk: Increased Insulin Sensitivity in Mice Lacking Protein Kinase C theta
Min Chen,
NIDDK, National Institutes of Health, USA
Short Talk: Glucose Intolerance Despite Normal Insulin Secretion and Responsiveness in Mice with Muscle-Specific Gs-alpha Deficiency
Short Talk: Glucose Intolerance Despite Normal Insulin Secretion and Responsiveness in Mice with Muscle-Specific Gs-alpha Deficiency
16:45—19:00
Metabolic Cross Talk (Joint)
Meeting has ended...abstracts no longer viewable online.
Gerald I. Shulman,
Yale University School of Medicine, USA
Unraveling the Mechanism of Insulin Resistance in Man: Potential Role of Mitochondrial Dysfunction
Unraveling the Mechanism of Insulin Resistance in Man: Potential Role of Mitochondrial Dysfunction
Katja A. Lamia,
The Scripps Research Institute, USA
Short Talk: p15P4KBeta Modulates Insulin Response
Short Talk: p15P4KBeta Modulates Insulin Response
*Session Chair †Invited, not yet responded.
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